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N1L Is an Ectromelia Virus Virulence Factor and Essential for In Vivo  Spread upon Respiratory Infection | Journal of Virology
N1L Is an Ectromelia Virus Virulence Factor and Essential for In Vivo Spread upon Respiratory Infection | Journal of Virology

Frontiers | The in Vitro Inhibitory Effect of Ectromelia Virus Infection on  Innate and Adaptive Immune Properties of GM-CSF-Derived Bone Marrow Cells  Is Mouse Strain-Independent | Microbiology
Frontiers | The in Vitro Inhibitory Effect of Ectromelia Virus Infection on Innate and Adaptive Immune Properties of GM-CSF-Derived Bone Marrow Cells Is Mouse Strain-Independent | Microbiology

Heterotypic immunity against vaccinia virus in an HLA-B*07:02 transgenic  mousepox infection model | Scientific Reports
Heterotypic immunity against vaccinia virus in an HLA-B*07:02 transgenic mousepox infection model | Scientific Reports

Ectromelia virus upregulates the expression of heat shock protein 70 to  promote viral replication
Ectromelia virus upregulates the expression of heat shock protein 70 to promote viral replication

Ectromelia virus - Wikipedia
Ectromelia virus - Wikipedia

Ectromelia Virus - Virology - Central Lakes Medical
Ectromelia Virus - Virology - Central Lakes Medical

PDF) Mousepox resulting from use of ectromelia virus-contaminated, imported  mouse serum
PDF) Mousepox resulting from use of ectromelia virus-contaminated, imported mouse serum

Mouse NK cell recognition of ectromelia virus-infected cells. The... |  Download Scientific Diagram
Mouse NK cell recognition of ectromelia virus-infected cells. The... | Download Scientific Diagram

Frontiers | The in Vitro Inhibitory Effect of Ectromelia Virus Infection on  Innate and Adaptive Immune Properties of GM-CSF-Derived Bone Marrow Cells  Is Mouse Strain-Independent | Microbiology
Frontiers | The in Vitro Inhibitory Effect of Ectromelia Virus Infection on Innate and Adaptive Immune Properties of GM-CSF-Derived Bone Marrow Cells Is Mouse Strain-Independent | Microbiology

Granzyme A is critical for recovery of mice from infection with the natural  cytopathic viral pathogen, ectromelia. - Abstract - Europe PMC
Granzyme A is critical for recovery of mice from infection with the natural cytopathic viral pathogen, ectromelia. - Abstract - Europe PMC

PDF) Hsp-27, hsp-70 and hsp-90 expression and apoptosis in macrophages  during ectromelia (mousepox) virus infection
PDF) Hsp-27, hsp-70 and hsp-90 expression and apoptosis in macrophages during ectromelia (mousepox) virus infection

Evidence for Persistence of Ectromelia Virus in Inbred Mice, Recrudescence  Following Immunosuppression and Transmission to Naïve Mice
Evidence for Persistence of Ectromelia Virus in Inbred Mice, Recrudescence Following Immunosuppression and Transmission to Naïve Mice

Ectromelia virus: the causative agent of mousepox | Microbiology Society
Ectromelia virus: the causative agent of mousepox | Microbiology Society

Pathogenesis and Immunity in Ectromelia Virus Infection of the Nasal Mucosa  of the Rat
Pathogenesis and Immunity in Ectromelia Virus Infection of the Nasal Mucosa of the Rat

PDF) Ectromelia Virus Disease Characterization in the BALB/c Mouse: A  Surrogate Model for Assessment of Smallpox Medical Countermeasures
PDF) Ectromelia Virus Disease Characterization in the BALB/c Mouse: A Surrogate Model for Assessment of Smallpox Medical Countermeasures

Ectromelia Virus (Mousepox)
Ectromelia Virus (Mousepox)

Viruses | Free Full-Text | Ectromelia Virus Disease Characterization in the  BALB/c Mouse: A Surrogate Model for Assessment of Smallpox Medical  Countermeasures | HTML
Viruses | Free Full-Text | Ectromelia Virus Disease Characterization in the BALB/c Mouse: A Surrogate Model for Assessment of Smallpox Medical Countermeasures | HTML

The Ectromelia Virus SPI-2 Protein Causes Lethal Mousepox by Preventing NK  Cell Responses | Journal of Virology
The Ectromelia Virus SPI-2 Protein Causes Lethal Mousepox by Preventing NK Cell Responses | Journal of Virology

Resistance to lethal ectromelia virus infection requires Type I interferon  receptor in natural killer cells and monocytes but not in adaptive immune  or parenchymal cells
Resistance to lethal ectromelia virus infection requires Type I interferon receptor in natural killer cells and monocytes but not in adaptive immune or parenchymal cells

Ectromelia virus lacking the E3L ortholog is replication-defective and  nonpathogenic but does induce protective immunity in a mouse strain  susceptible to lethal mousepox - ScienceDirect
Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox - ScienceDirect

Ectromelia virus lacking the E3L ortholog is replication-defective and  nonpathogenic but does induce protective immunity in a mouse strain  susceptible to lethal mousepox - ScienceDirect
Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox - ScienceDirect

Resistance to lethal ectromelia virus infection requires Type I interferon  receptor in natural killer cells and monocytes but not in adaptive immune  or parenchymal cells
Resistance to lethal ectromelia virus infection requires Type I interferon receptor in natural killer cells and monocytes but not in adaptive immune or parenchymal cells

Ectromelia virus lacking the E3L ortholog is replication-defective and  nonpathogenic but does induce protective immunity in a mouse strain  susceptible to lethal mousepox - ScienceDirect
Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox - ScienceDirect

Viruses | Free Full-Text | Ectromelia Virus Disease Characterization in the  BALB/c Mouse: A Surrogate Model for Assessment of Smallpox Medical  Countermeasures | HTML
Viruses | Free Full-Text | Ectromelia Virus Disease Characterization in the BALB/c Mouse: A Surrogate Model for Assessment of Smallpox Medical Countermeasures | HTML

Ectromelia virus lacking the E3L ortholog is replication-defective and  nonpathogenic but does induce protective immunity in a mouse strain  susceptible to lethal mousepox - ScienceDirect
Ectromelia virus lacking the E3L ortholog is replication-defective and nonpathogenic but does induce protective immunity in a mouse strain susceptible to lethal mousepox - ScienceDirect